Non lethal Inhibition of Gut Microbial Trimethylamine Production for the Treatment of Atherosclerosis: Cell Gut microbe derived metabolite trimethylamine N oxide activates PERK to drive fibrogenic mesenchymal differentiation: iScience TMAO (Trimethylamine oxide) biocrates life science L Carnitine intake and high trimethylamine N oxide plasma levels correlate with low aortic lesions in ApoE transgenic mice expressing CETP Atherosclerosis Intestinal microbiota metabolism of l carnitine, a nutrient in red meat, promotes atherosclerosis Nature Medicine